References

  1. Okubo Y et al. A D variant, Del? Transfusion, 1984. [Citation] [RHeference]
  2. Omi T et al. The genomic organization of the partial D category DVa: the presence of a new partial D associated with the DVa phenotype. Biochem Biophys Res Commun, 1999. [Citation] [RHeference]
  3. Omi T et al. Detection of Rh23 in the partial D phenotype associated with the D(Va) category. Transfusion, 2000. [Citation] [RHeference]
  4. Okuda H et al. The analysis of nucleotide substitutions, gaps, and recombination events between RHD and RHCE genes through complete sequencing. Biochem Biophys Res Commun, 2000. [Citation] [RHeference]
  5. Kashiwase K et al. E variants found in Japanese and c antigenicity alteration without substitution in the second extracellular loop. Transfusion, 2001. [Citation] [RHeference]
  6. Kamesaki T et al. Molecular characterization of weak D phenotypes by site-directed mutagenesis and expression of mutant Rh-green fluorescence protein fusions in K562 cells. Vox Sang, 2001. [Citation] [RHeference]
  7. Omi T et al. Isolation, characterization, and family study of DTI, a novel partial D phenotype affecting the fourth external loop of D polypeptides. Transfusion, 2002. [Citation] [RHeference]
  8. Kamesaki T et al. A novel mutation in the RHD gene in Japanese individuals with weak D, encoding an amino acid change in the 11th transmembranous domain of the RhD protein. Vox Sang, 2003. [Citation] [RHeference]
  9. L. Tilley et al. Four RhD Variants Caused by Novel RhD Mutations Transfusion Medicine, 2007. — Abstract — [RHeference]