References
- Okuda H et al. The RHD gene is highly detectable in RhD-negative Japanese donors. J Clin Invest, 1997. [Citation] [RHeference]
- Chang JG et al. Human RhDel is caused by a deletion of 1,013 bp between introns 8 and 9 including exon 9 of RHD gene. Blood, 1998. [Citation] [RHeference]
- Xu Q et al. [Study on polymorphism of D gene exons among RhD-negative Chinese Han population]. Zhonghua Yi Xue Yi Chuan Xue Za Zhi, 2001. [Citation] [RHeference]
- Lee YL et al. Analysis of RHD genes in Taiwanese RhD-negative donors by the multiplex PCR method. J Clin Lab Anal, 2003. [Citation] [RHeference]
- Chen JC et al. RHD 1227A is an important genetic marker for RhD(el) individuals. Am J Clin Pathol, 2004. [Citation] [RHeference]
- Shao C et al. Whole exon 5 and intron 5 replaced by RHCE in DVa(Hus). J Hum Genet, 2004. [Citation] [RHeference]
- Li Q et al. Molecular basis of D variants between Uigur and Han blood donors in Xinjiang. Transfus Med, 2008. [Citation] [RHeference]
- Liu HC et al. Aberrant RNA splicing in RHD 7-9 exons of DEL individuals in Taiwan: a mechanism study. Biochim Biophys Acta, 2010. [Citation] [RHeference]
- Xu H et al. [Molecular study of two novel RHD alleles and pedigree analysis]. Zhonghua Yi Xue Yi Chuan Xue Za Zhi, 2011. [Citation] [RHeference]
- Yukari Nishiyama et al. A case of suspected partial D because of weak reactivity to anti-D on column agglutination technology and identified as partial D (DBT-1) by genetic testing Japanese Journal of Transfusion and Cell Therapy, 2011. — Article — [RHeference]
- Shao CP et al. DEL RBC transfusion should be avoided in particular blood recipient in East Asia due to allosensitization and ineffectiveness. J Zhejiang Univ Sci B, 2012. [Citation] [RHeference]
- Ye L et al. Partial D phenotypes and genotypes in the Chinese population. Transfusion, 2012. [Citation] [RHeference]
- Ye SH et al. A comprehensive investigation of RHD polymorphisms in the Chinese Han population in Xi'an. Blood Transfus, 2014. [Citation] [RHeference]
- Gu J et al. Analysis of density and epitopes of D antigen on the surface of erythrocytes from DEL phenotypic individuals carrying the RHD1227A allele. Blood Transfus, 2014. [Citation] [RHeference]
- Gu J et al. Molecular basis of DEL phenotype in the Chinese population. BMC Med Genet, 2014. [Citation] [RHeference]
- Crottet SL et al. Implementation of a mandatory donor RHD screening in Switzerland. Transfus Apher Sci, 2014. [Citation] [RHeference]
- Wang QP et al. An investigation of secondary anti-D immunisation among phenotypically RhD-negative individuals in the Chinese population. Blood Transfus, 2014. [Citation] [RHeference]
- Wang M et al. Anti-D alloimmunisation in pregnant women with DEL phenotype in China. Transfus Med, 2015. [Citation] [RHeference]
- Xu W et al. Prospective Evaluation of a Transfusion Policy of RhD-Positive Red Blood Cells into DEL Patients in China. Transfus Med Hemother, 2015. [Citation] [RHeference]
- Chen DP et al. Comprehensive analysis of RHD splicing transcripts reveals the molecular basis for the weak anti-D reactivity of Del -red blood cells. Transfus Med, 2016. [Citation] [RHeference]
- Lopez GH et al. A D+ blood donor with a novel RHD*D-CE(5-6)-D gene variant exhibits the low-frequency antigen RH23 (D(W) ) characteristic of the partial DVa phenotype. Transfusion, 2016. [Citation] [RHeference]
- Hyland CA et al. Non-invasive fetal RHD genotyping for RhD negative women stratified into RHD gene deletion or variant groups: comparative accuracy using two blood collection tube types. Pathology, 2017. [Citation] [RHeference]
- Ji YL et al. RHD genotype and zygosity analysis in the Chinese Southern Han D+, D- and D variant donors using the multiplex ligation-dependent probe amplification assay. Vox Sang, 2017. [Citation] [RHeference]
- McGowan EC et al. Diverse and novel RHD variants in Australian blood donors with a weak D phenotype: implication for transfusion management. Vox Sang, 2017. [Citation] [RHeference]
- Lopez GH et al. Genotyping by sequencing defines independent novel RHD variants for an antenatal patient and a blood donor. Transfusion, 2017. [Citation] [RHeference]
- Luo H et al. [Phenotype Types and Genetic Mutation Mechanism of Rhesus D Variant Individuals]. Zhongguo Shi Yan Xue Ye Xue Za Zhi, 2017. [Citation] [RHeference]
- Turley E et al. Severe hemolytic disease of the fetus and newborn due to allo-anti-D in a patient with a partial DEL phenotype arising from the variant allele described as RHD*148+1T (RHD*01EL.31). Transfusion, 2018. [Citation] [RHeference]
- Lopez GH et al. A DEL phenotype attributed to RHD Exon 9 sequence deletion: slipped-strand mispairing and blood group polymorphisms. Transfusion, 2018. [Citation] [RHeference]
- Schoeman EM et al. Targeted exome sequencing defines novel and rare variants in complex blood group serology cases for a red blood cell reference laboratory setting. Transfusion, 2018. [Citation] [RHeference]
- Liao Z et al. [A weak D type 59 case identified in the Chinese Han population]. Zhonghua Yi Xue Yi Chuan Xue Za Zhi, 2018. [Citation] [RHeference]
- Xu HY et al. [Identification of Phenotype and Genotype in One Case with Weak D blood group]. Zhongguo Shi Yan Xue Ye Xue Za Zhi, 2018. [Citation] [RHeference]
- Qian C et al. A new RhD variant allele is caused by a RhD 26 T > G mutation in a Chinese Han woman with a weak D phenotype. Transfusion, 2019. [Citation] [RHeference]