References
- Lomas C et al. Demonstration of seven epitopes on the Rh antigen D using human monoclonal anti-D antibodies and red cells from D categories. Vox Sang, 1989. [Citation] [RHeference]
- Mayne K et al. Rh immunization by the partial D antigen of category DVa. Br J Haematol, 1990. [Citation] [RHeference]
- Okubo Y et al. Partial D antigens disclosed by a monoclonal anti-D in Japanese blood donors. Transfusion, 1991. [Citation] [RHeference]
- Lomas C et al. Further complexities of the Rh antigen D disclosed by testing category DII cells with monoclonal anti-D. Transfus Med, 1993. [Citation] [RHeference]
- Lomas C et al. Abolition of the DVc subcategory. Transfusion, 1993. [Citation] [RHeference]
- Lomas C et al. FPTT is a low-incidence Rh antigen associated with a "new" partial Rh D phenotype, DFR. Transfusion, 1994. [Citation] [RHeference]
- Wallace M et al. The D antigen characteristic of RoHar is a partial D antigen. Vox Sang, 1996. [Citation] [RHeference]
- Tippett P et al. The Rh antigen D: partial D antigens and associated low incidence antigens. Vox Sang, 1996. [Citation] [RHeference]
- Wallace M et al. DBT: a partial D phenotype associated with the low-incidence antigen Rh32. Transfus Med, 1997. [Citation] [RHeference]
- Reid ME et al. Two examples of an inseparable antibody that reacts equally well with DW+ and Rh32+ red blood cells. Vox Sang, 1998. [Citation] [RHeference]
- Reid ME et al. DAK, a new low-incidence antigen in the Rh blood group system. Transfusion, 2003. [Citation] [RHeference]
- Westhoff CM et al. A new hybrid RHCE gene (CeNR) is responsible for expression of a novel antigen. Transfusion, 2004. [Citation] [RHeference]
- Lomas-Francis C et al. A confusion in antibody identification: anti-D production after anti-hrB. Immunohematology, 2007. [Citation] [RHeference]
- Westhoff CM et al. DIIIa and DIII Type 5 are encoded by the same allele and are associated with altered RHCE*ce alleles: clinical implications. Transfusion, 2010. [Citation] [RHeference]
- Hipsky CH et al. RHCE*ceCF encodes partial c and partial e but not CELO, an antigen antithetical to Crawford. Transfusion, 2011. [Citation] [RHeference]
- S Vege et al. Weak D Type 2 and Production of Anti-D Transfusion, 2011. — Abstract — [RHeference]
- C Lomas-Francis et al. Surprising findings with RBCs expressing the low prevalence RH antigen Evans Transfusion, 2011. — Abstract — [RHeference]
- Hue-Roye K et al. Red cells from the original JAL+ proband are also DAK+ and STEM+. Vox Sang, 2011. [Citation] [RHeference]
- Hipsky CH et al. Molecular basis of the rare gene complex, DIVa(C)-, which encodes four low-prevalence antigens in the Rh blood group system. Vox Sang, 2012. [Citation] [RHeference]
- C Fong et al. An RHD*455C Allele Encodes a Partial D Phenotype Associated With Production of Allo Anti-D Transfusion, 2012. — Abstract — [RHeference]
- Reid ME et al. Molecular background of RH in Bastiaan, the RH:-31,-34 index case, and two novel RHD alleles. Immunohematology, 2012. [Citation] [RHeference]
- Lomas-Francis C et al. DIII Type 7 is likely the original serologically defined DIIIb. Transfusion, 2012. [Citation] [RHeference]
- Reid ME et al. The low-prevalence Rh antigen STEM (RH49) is encoded by two different RHCE*ce818T alleles that are often in cis to RHD*DOL. Transfusion, 2013. [Citation] [RHeference]
- Westhoff CM et al. RHCE*ceTI encodes partial c and partial e and is often in cis to RHD*DIVa. Transfusion, 2013. [Citation] [RHeference]
- Westhoff CM et al. RHCE*ceMO is frequently in cis to RHD*DAU0 and encodes a hr(S) -, hr(B) -, RH:-61 phenotype in black persons: clinical significance. Transfusion, 2013. [Citation] [RHeference]
- C M Westhoff et al. A Weak D Phenotype Discovered in Nine Patients in Western Canada due to a Nucleotide 1187C>G (Pro396Arg) Change in RHD Transfusion, 2014. — Abstract — [RHeference]
- G Shakarian et al. Three New, Clinically Relevant RHD genes Transfusion, 2014. — Abstract — [RHeference]
- Vege S et al. D typing discrepancies and anti-D production associated with six new RHD alleles. Transfusion, 2016. — Abstract — [RHeference]
- S Vege et al. Expansion of the RHD DAU Cluster: Identification of Two Additional Novel Alleles Transfusion, 2017. — Abstract — [RHeference]
- S Vege et al. Serologic Characterization of D Antigen Expression Encoded By Two Reported RHD Alleles: Implications for Transfusion and Pregnancy Transfusion, 2017. — Abstract — [RHeference]
- S Vege et al. RHD Genotyping of Discrepant or Weak D Samples: Over a Year’s Experience. Transfusion, 2017. — Abstract — [RHeference]
- J Aeschlimann et al. Gene Conversion within the r’S Haplotype Complicates RHD Genotype Interpretations Transfusion, 2017. — Abstract — [RHeference]
- J Aeschlimann et al. Serological and Molecular Characterization of Three New RHD Alleles Transfusion, 2018. — Abstract — [RHeference]
- J S. Woo et al. Robust Allo-Anti-D with Subsequent Anti-K Production after Transfusion of D-Positive RBCs to a Patient with Weak D Type 1 Transfusion, 2018. — Abstract — [RHeference]
- GA Denomme et al. A novel weak D 4.0-related allele and RHCE*ceCF defines a new rh haplotype in an autologous donor with an antibody to a high frequency antigen Vox Sanguinis, 2019. — Abstract — [RHeference]
- Sunitha Vege et al. Novel RHD Alleles Identified When Resolving Serologic Weak D Phenotypes Transfusion, 2019. — Abstract — [RHeference]
- Westhoff CM et al. Experience with RHD*weak D type 4.0 in the USA. Blood Transfus, 2019. [Citation] [RHeference]
- Mathur G et al. A novel RHD*DAU allele with c.1136C>T (p.Thr379Met) and c.17C>T (p. Pro6Leu). Transfusion, 2020. [Citation] [RHeference]
- Sunitha Vege et al. Novel RHD Alleles and Predicting Risk for Anti-D Transfusion, 2020. — Abstract — [RHeference]
- Sunitha Vege et al. Novel RHCE*ceS with c.462G>T (p. Arg154Ser) Encodes Expression of a D Reactive Epitope Transfusion, 2020. — Abstract — [RHeference]
- Vege S et al. Impact of RHD genotyping on transfusion practice in Denmark and the United States and identification of novel RHD alleles. Transfusion, 2021. [Citation] [RHeference]