References

  1. Huang CH et al. Identification of a partial internal deletion in the RH locus causing the human erythrocyte D--phenotype. Blood, 1995. [Citation] [RHeference]
  2. Huang CH et al. Human D(IIIa) erythrocytes: RhD protein is associated with multiple dispersed amino acid variations. Am J Hematol, 1997. [Citation] [RHeference]
  3. Reid ME et al. Use of LOR-15C9 monoclonal anti-D to differentiate erythrocytes with the partial DvI antigen from those with either partial D antigens or weak D antigens. Immunohematology, 1998. [Citation] [RHeference]
  4. Reid ME et al. Two examples of an inseparable antibody that reacts equally well with DW+ and Rh32+ red blood cells. Vox Sang, 1998. [Citation] [RHeference]
  5. Huang CH et al. Evidence for a separate genetic origin of the partial D phenotype DBT in a Japanese family. Transfusion, 1999. [Citation] [RHeference]
  6. Avent ND et al. The Rh blood group system: a review. Blood, 2000. [Citation] [RHeference]
  7. Reid ME et al. DAK, a new low-incidence antigen in the Rh blood group system. Transfusion, 2003. [Citation] [RHeference]
  8. Westhoff CM et al. A new hybrid RHCE gene (CeNR) is responsible for expression of a novel antigen. Transfusion, 2004. [Citation] [RHeference]
  9. Lomas-Francis C et al. A confusion in antibody identification: anti-D production after anti-hrB. Immunohematology, 2007. [Citation] [RHeference]
  10. Westhoff CM et al. DIIIa and DIII Type 5 are encoded by the same allele and are associated with altered RHCE*ce alleles: clinical implications. Transfusion, 2010. [Citation] [RHeference]
  11. Hipsky CH et al. RHCE*ceCF encodes partial c and partial e but not CELO, an antigen antithetical to Crawford. Transfusion, 2011. [Citation] [RHeference]
  12. Hue-Roye K et al. Red cells from the original JAL+ proband are also DAK+ and STEM+. Vox Sang, 2011. [Citation] [RHeference]
  13. Hipsky CH et al. Molecular basis of the rare gene complex, DIVa(C)-, which encodes four low-prevalence antigens in the Rh blood group system. Vox Sang, 2012. [Citation] [RHeference]
  14. M E Reid et al. DIVa and DIVa-2 are Encoded by the Same RHD Allele Transfusion, 2012. — Abstract — [RHeference]
  15. Reid ME et al. Molecular background of RH in Bastiaan, the RH:-31,-34 index case, and two novel RHD alleles. Immunohematology, 2012. [Citation] [RHeference]
  16. Lomas-Francis C et al. DIII Type 7 is likely the original serologically defined DIIIb. Transfusion, 2012. [Citation] [RHeference]
  17. Reid ME et al. The low-prevalence Rh antigen STEM (RH49) is encoded by two different RHCE*ce818T alleles that are often in cis to RHD*DOL. Transfusion, 2013. [Citation] [RHeference]
  18. Westhoff CM et al. RHCE*ceTI encodes partial c and partial e and is often in cis to RHD*DIVa. Transfusion, 2013. [Citation] [RHeference]
  19. Westhoff CM et al. RHCE*ceMO is frequently in cis to RHD*DAU0 and encodes a hr(S) -, hr(B) -, RH:-61 phenotype in black persons: clinical significance. Transfusion, 2013. [Citation] [RHeference]
  20. Reid ME et al. Genomic analyses of RH alleles to improve transfusion therapy in patients with sickle cell disease. Blood Cells Mol Dis, 2014. [Citation] [RHeference]